Which of the following answers about thrombosis therapy is false?
1. Radiation therapy could be develop of SVC Thrombosis
2. Chemotherapy could be develop of SVC Thrombosis
3. The main catheters in SVC could develop of thrombosis
4. the SVC thrombosis deriving from complications related to the positioning of the central venous catherter
5. These collateral pathways bypassed the obstruction, provided venous drainage from the head, neck, chest, and upper extremities

The correct answers for the question "Which of the following answers about thrombosis therapy is false?" are:

1. Radiation therapy could be develop of SVC Thrombosis

2. Chemotherapy could be develop of SVC Thrombosis

4. the SVC thrombosis deriving from complications related to the positioning of the central venous catherter

5. These collateral pathways bypassed the obstruction, provided venous drainage from the head, neck, chest, and upper extremities

1. Patients with Hodgkin lymphoma and radiation therapy could develop thrombosis (at the beginning or at the end of the treatment)

2. Patients with Hodgkin lymphoma could develop thrombosis (infusion of chemotherapeutic agents may cause the progressive degeneration of the vessel wall and the exposure of the endothelium and the development of thrombosis).

3.The thrombosis in SVC is more often present with medium-long term catheter ( Porth a cath )

4.The positioning of the central venous catherter could cause thrombosis (It`s often present during follow up therapy)

5.Collateral vessels provided venous drainage.(this is only mechanism for asyntomatic patients).



From the manuscript:
Compensatory dilatation of the Azygos Venous system Secondary To Superior Vena Cava Occlusion
Radiology Case. 2009 Dec; 3(12):49-55


This article belongs to the Chest section.
From the manuscript

Compensatory dilatation of the Azygos Venous system Secondary To Superior Vena Cava Occlusion

Abstract

Compensatory dilatation of the Azygos Venous system Secondary To Superior Vena Cava Occlusion Superior vena cava (SVC) occlusion can be clinically recognized in the acute setting when the stenosing process does not allow the development of collateral venous channels, which guarantee the venous drainage to the right heart. On the contrary, when the obstruction develops progressively, the diagnosis of SVC obstruction may remain undiagnosed. In the present case, the presence of SVC thrombosis was purely coincidental. In fact, the obstruction was first noticed on diagnostic tests performed because of the malfunction of a totally implantable Porth a Cath placed into the superior vena cava (through right subclavian access), five years before, in a patient suffering from non-Hodgkin disease. Venography is the most appropriate diagnostic methodology which reveals the presence of a dilated azygos vein as a compensatory mechanism. Comparison with computed tomography allows to confirm the diagnosis and to identify the possible causes. Dilatation of the azygos vein, secondary to superior vena cava thrombosis, although a rare event, should be taken into consideration in those patients with CVC and who present with frequent episodes of deep venous thrombosis.

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